The early onset of diabetes during childhood, particularly type 2 diabetes, is becoming a growing healthcare concern in the United States. Currently, nearly 100 million Americans are estimated to have diabetes or are believed to be at extreme risk for developing diabetes (prediabetes). Additionally, an estimated 132,000 of these cases have been diagnosed in children and adolescents younger than 18 (Centers for Disease Control and Prevention, 2017).
Historically, when children were diagnosed with diabetes, it was most often insulin-dependent type 1 diabetes, which is caused by an autoimmune disease that shuts down the body’s ability to make insulin. By contrast, type 2 diabetes has traditionally been termed “adult-onset” because its prevalence is highly impacted by diet, activity levels and lifestyle-related factors.
Currently, type 2 diabetes is growing at a rate of roughly 5,000 new cases per year in youth and adolescents (Lawrence et al., 2014), and the diagnosis of type 2 diabetes in this population has increased more than 30% in the last 10 years (Dabelea et al., 2014).
It appears that the increasing rates of type 2 diabetes in children could be yet another health concern stemming from the broader childhood obesity epidemic. Physical inactivity, poor diet and excess body fat have been identified as risk factors for type 2 diabetes in both adults and children (Copeland, 2011). Children who are in the 85th percentile or higher for BMI are more than four times more likely to develop type 2 diabetes (Abbasi et al., 2017). Currently, this represents roughly one in three children (Ogden et al., 2014).
The Mechanisms Behind Type 2 Diabetes
One of the primary mechanisms behind type 2 diabetes appears to be a decrease in insulin sensitivity (Hannon and Arslanian, 2015). When tissues and cells lose sensitivity to insulin, the result is damage to the metabolic infrastructure that helps regulate blood sugar levels. When elevated blood sugar becomes chronic, it creates a variety of acute and longitudinal medical complications. The risk factors listed in the paragraph above significantly contribute to a reduction in insulin sensitivity (Copeland et al., 2011).
Medical complications stemming from type 2 diabetes include microalbuminuria, hypertension, kidney disease, dyslipidemia, and neuropathy. Advanced levels of these complications generally appear about 10 years from diagnosis (Hannon, Arslanian, 2015). This could significantly increase risks for morbidity and mortality during the most productive years of life when kids grow up to join the workforce and have families. It also prolongs the physical and financial toll brought on by the disease.
How to Reduce a Child’s Risk of Developing Type 2 Diabetes
Clearly, an increase in the prevalence of type 2 diabetes in children has future implications for the nation’s workforce and healthcare system, as well as individual health and happiness. The good news is that through basic lifestyle modification, a child’s risk of developing type 2 diabetes and the associated health complications can be significantly decreased.
To turn the tide on this disturbing trend, a variety of recommendations are provided by the Centers for Disease Control and Prevention (2017):
Replhealthsite the consumption of sugary drinks with water
Eat more fruits and vegetables
Involve children in the preparation of healthy meals
Eat meals at the table instead of in front of the television or individually in a room
Teach kids about food labels
Eat together as a family
Serve smaller portions
Facilitate 60 minutes of activity daily
Involve children in sports
It’s also recommended that parents of talk to their pediatricians about the risks for developing type 2 diabetes, so early action can halt progression of the disease.
When we take steps to create a culture of wellness at home by supporting and exhibiting healthy habits, we can increase the likelihood that our children will grow up to become happy and healthy adults.
Abbasi, A., et al. (2017). Body mass index and incident type 1 and type 2 diabetes in children and young adults: A retrospective cohort study. Journal of the Endocrine Society, 1, 5, 524-537.
Centers for Disease Control and Prevention (2017). National Diabetes Statistics Report, 2017. Atlanta, Ga.: Centers for Disease Control and Prevention, U.S. Dept of Health and Human Services.
Copeland, K.C. et al. (2011). Characteristics of adolescents and youth with recent-onset type 2 diabetes: The TODAY cohort at baseline. The Journal of Clinical Endocrinology and Metabolism, 96, 1, 159–167.
Dabelea, D. et al. (2014). Prevalence of type 1 and type 2 diabetes among children and adolescents from 2001 to 2009. Journal of the American Medical Association, 311, 17, 1778–1786.
Hamman, R.F. et al. (2014). The SEARCH for Diabetes in Youth study: Rationale, findings, and future directions. Diabetes care, 37, 12, 3336–3344.
Hannon, T.S. and Arslanian, S.A. (2015). The changing fhealthsite of diabetes in youth: Lessons learned from studies of type 2 diabetes. Annals of the New York Academy of Sciences, 1353, 113-137.
Lawrence, J.M. et al. (2014). Incidence of diabetes in U.S. youth by type, rhealthsite/ethnicity and age, 2008–2009. Diabetes, 63, (Supplement 1), A407.
Ogden, C.L. et al. (2014). Prevalence of childhood and adult obesity in the United States, 2011-2012. Journal of the American Medical Association, 311, 8, 806–814.